Name (Synonyms) | Correlation | |
---|---|---|
drug410 | Biological data Wiki | 0.82 |
drug792 | Covid19 Wiki | 0.58 |
Name (Synonyms) | Correlation | |
---|---|---|
D007238 | Infarction NIH | 0.44 |
D009203 | Myocardial Ischemia NIH | 0.33 |
D009205 | Myocarditis NIH | 0.20 |
D006973 | Hypertension NIH | 0.15 |
D002318 | Cardiovascular Diseases NIH | 0.11 |
Name (Synonyms) | Correlation | |
---|---|---|
HP:0001658 | Myocardial infarction HPO | 0.33 |
HP:0012819 | Myocarditis HPO | 0.20 |
HP:0000822 | Hypertension HPO | 0.15 |
HP:0001626 | Abnormality of the cardiovascular system HPO | 0.11 |
There are 3 clinical trials
Hospitalized patients with COVID-19 will be included in the study in centers around Poland. After the hospitalization, a short questionnaire will be completed, including pre-hospitalization diagnoses, pre-hospitalization medications, clinical status on admission, the course, complication and the duration of hospitalization. The questionnaire will be available in paper form and on-line.
Description: Death, myocardial infarction, heart failure, myocarditis, acute renal failure, stroke
Measure: Adverse events Time: through study completion, an average of 2 weeksDescription: Number of participants with death, myocardial infarction, heart failure, myocarditis, acute renal failure, stroke analyzed separately for each of the endpoints
Measure: Number of participants with death, myocardial infarction, heart failure, myocarditis, acute renal failure, stroke Time: through study completion, an average of 2 weeksDescription: Ventilation during hospitalization
Measure: Ventilation during hospitalization Time: through study completion, an average of 2 weeksDescription: Number of participants with death, myocardial infarction, heart failure, myocarditis, acute renal failure, stroke analyzed separately for each of the endpoints
Measure: Number of participants with death, myocardial infarction, heart failure, myocarditis, acute renal failure, stroke Time: prolonged follow up, through study completion, an average of one yearMyocardial injury, as assessed by elevation of cardiac troponins (Tnc), is frequent among patients with COVID-19. Although rare autopsy cases reported COVID-19 related myocardial inflammation, the origin of Tnc elevation is unknown to date. Several cardiac causes, such as myocarditis, non-ischemic myocardial injury (NIMI), or myocardial infarction (MI) may lead to Tnc kinetic. Our work will test the hypothesis that during SARS-Cov2 infection, the elevation of cardiac biomarkers could be linked to the occurrence of myocarditis.
Description: Myocardtitis diagnosis in patients COVID+ and troponin+
Measure: characterize the myocardial damage associated with CoV-2 SARS infection Time: Through study completion, an average of 1 yearThe COVID-19 pandemic highlights the importance of the prognosis of co-morbidities, such as coronary artery disease, which significantly increase the risk of mortality in patients infected with SARS-CoV2. Investigators have recently studied the complex links between respiratory infections, particularly pneumonia, and type 2 myocardial infarction (MI) in many respects. The etiology of type 2 MI is based on an imbalance of myocardial oxygen supply/need in the absence of rupture/erosion of atheromatous plaques. Based on the RICO survey data, the investigators investigated whether COVID-19-related sepsis and/or respiratory failure could be an underlying mechanism of MI2.